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Otherwise, Nfkb1 , as the central regulator of neuroinflammation‐associated disease pathogenesis (Niranjan, 2013), induces reactive oxygen species and proinflammatory cytokines (such as IL‐1β, interferon‐γ, and TNF‐α) that cause secondary neurotoxicity (Block, Zecca, & Hong, 2007

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Patients with vitiligo have lower total serum antioxidant status, a higher oxidative stress index and total serum oxidant status in both generalised and localised vitiligo patients