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When IFN-γ binds to its receptor, the Janus kinase (JAK)/signal transducers and activators of transcription (STAT) pathway is activated and stimulates the transcription of the chemokine ligands CXCL9 and CXCL10 [53]
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The rapid release of IL-4 is then followed by production of IFN-γ, acting as a negative regulator by promoting apoptosis of neutrophils (67)
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