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substantiated that APS treatment markedly attenuated the migration and invasiveness of non-small-cell lung cancer (NSCLC) cells compared to that seen with the control, and the underlying mechanism may be related to the APS-related increase in cellular miR-195-5p levels (109)

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Determining the signaling pathways and molecular players that regulate NET release is therefore critical, not only for advancing our understanding of neutrophil biology but also for identifying new therapeutic targets to treat disorders associated with aberrant NETosis