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The study also found that myricetin treatment can induce autophagy by influencing ER stress and unfolded protein response (UPR) signaling pathways, such as serine/threonine-protein kinase/endoribonuclease inositol-requiring enzyme 1 α-c-Jun N-terminal kinases (IRE1α-JNK) and Ca2+- AMP-activated protein kinase (AMPK), which may act as upstream regulators of the mTOR
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