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The following is a summary of documented tissue systems from peer-reviewed literature: This cross-tissue activity profile is directly attributable to the fact that its primary mechanisms — VEGFR2-driven angiogenesis and NO system modulation — are not tissue-specific
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(B) Oxidative stress marker (MDA) and activities of antioxidant enzymes (Catalase and Glutathione reductase-GR) were measured in LP1 cells lentivirally transduced with pLVempty vector or pLV CD38 OE
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