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UA treatment modulates the KLF2-VEGF-A axis and improves the integrity of the endothelial cell barrier, leading to neuroprotection (Fig
Cordyceps
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They influence the initiation, progression, and resolution of neuroinflammation through multiple mechanisms, including the release of inflammatory mediators, regulation of immune cell infiltration, interactions with microglia and endothelial cells, inflammasome activation, extracellular matrix degradation, and phenotypic plasticity (Tastan and Heneka, 2024
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